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Shaping the macrophage landscape in the tumour microenvironment
 
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1
Department of Human Immunology, Institute of Medical Sciences, University of Rzeszow, Rzeszow, Poland
 
2
Institute of Biology, University of Szczecin, Szczecin, Poland
 
3
Centre for Experimental Immunology and Immunobiology in Infectious Diseases and Cancer, University of Szczecin, Szczecin, Poland
 
4
Centre of Regenerative Medicine, Medical University of Bialystok, Bialystok, Poland
 
 
Publication date: 2025-12-10
 
 
Cent Eur J Immunol 2025;(3):233
 
REFERENCES (8)
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Lu J, Ge C, Yu P, et al. (2025): LAMA1 derived from colorectal cancer promotes M2 polarization in macrophages via activation of the EGFR/AKT/CREB pathway. Cent Eur J Immunol 50: 234-247.
 
2.
Huang L, Duan F, Dong X, Zhang Z (2024): The N6-methyladenosine pattern of MAP3K7 mediates the effects of sevoflurane on macrophage M2 polarization and cervical cancer migration and invasion. Cent Eur J Immunol 49: 393-403.
 
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Zhou L, Ma D, Li X, et al. (2025): Macrophage M2 polarization induced by ANKRD22 in lung adenocarcinoma facilitates tumor angiogenesis. Cent Eur J Immunol 50: 38-51.
 
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Zhang L, Zhang K, Zhang J, et al. (2021): Loss of fragile site-associated tumor suppressor promotes antitumor immunity via macrophage polarization. Nat Commun 12: 4300.
 
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Wang Q, Wu Y, Long Y, et al. (2025): AR+TREM2+ macrophage induced pathogenic immunosuppression promotes prostate cancer progression. Nat Commun 16: 6964.
 
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Ramos RN, Rodriguez C, Hubert M, et al. (2020): CD163+ tumor-associated macrophage accumulation in breast cancer patients reflects both local differentiation signals and systemic skewing of monocytes. Clin Transl Immunology 9: e1108.
 
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Im JH, Buzzelli JN, Jones K, et al. (2020): FGF2 alters macrophage polarization, tumour immunity and growth and can be targeted during radiotherapy. Nat Commun 11: 4064.
 
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Casanova-Acebes M, Dalla E, Leader AM, et al. (2021): Tissue-resident macrophages provide a pro-tumorigenic niche to early NSCLC cells. Nature 595: 578-584.
 
eISSN:1644-4124
ISSN:1426-3912
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